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Preliminary experiments reveal conolidine may inhibit particular ion channels, cutting down neuronal excitability and restricting suffering signals. This mechanism is particularly appropriate in neuropathic ache, where irregular signaling brings about persistent soreness. Additionally, conolidine appears to impact G protein-coupled receptor (GPCR)
Preliminary scientific studies point out conolidine may inhibit certain ion channels, lessening neuronal excitability and limiting ache indicators. This system is especially pertinent in neuropathic agony, where abnormal signaling brings about persistent pain. Moreover, conolidine seems to influence G protein-coupled receptor (GPCR) pathways integr
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